Schizofrenia a zmiany w mózgu

[fuKs]

No dobrze, ale jaka alternatywa?

Załóżmy sektę nie leczących się, zamieszkamy na jednym osiedlu.
Wieczorem grill, a w nocy schiz-party.

Nawet sobie tego nie wyobrażam

[aux03ster03]

bedziemy sie wymieniac urojeniami ;

[Unknown]

"Brain damage, dementia, and persistent cognitive dysfunction associated with neuroleptic drugs: evidence, etiology, implications." - Peter R. Breggin, Center for the Study of Psychiatry and George Mason University: Istnieją przekonujące dowody, które wskazują na to, że długotrwałe stosowanie neuroleptyków powoduje upośledzenie funkcji poznawczych, otępienie i atrofię najwyższych ośrodków mózgu. Ponadto potwierdzono, iż neuroleptyki mogą powodować reaktywne późne psychozy. Nie ma dowodów na to, że schizofrenia powoduje któreś z tych skutków ubocznych, a tym bardziej na wpływ jej samej na demencje czy atrofie mózgu.

[moi]

Nie ma dowodów na to, że schizofrenia powoduje któreś z tych skutków ubocznych, a tym bardziej na wpływ jej samej na demencje czy atrofie mózgu.

Są za to dowody na to, że psychoza potrafi całkowicie wyeliminować człowieka z życia społecznego, rodzinnego, jakiegokolwiek...

Pozdrawiam.m.

[Unknown]

Są za to dowody na to, że psychoza potrafi całkowicie wyeliminować człowieka z życia społecznego, rodzinnego, jakiegokolwiek.

Czy ktoś twierdził inaczej?

[moi]

Nie, po prostu warto o tym pamiętać robiąc bilans zysków i strat związany z farmakoterapią.

Pozdrawiam.m.

[Unknown]

"Brain damage, dementia, and persistent cognitive dysfunction associated with neuroleptic drugs: evidence, etiology, implications." - Peter R. Breggin, Center for the Study of Psychiatry and George Mason University
ENG: http://www.breggin.com/index.php?option ... d=99999999
GER: http://www.breggin.com/index.php?option ... d=99999999

[Unknown]

Relapse & Antipsychotic Treatment in Schizophrenia Change Brain...
http://bbrfoundation.org/brain-matters- ... ange-brain
Relapse Duration, Treatment Intensity, and Brain Tissue Loss in Schizophrenia: A Prospective Longitudinal MRI Study http://ajp.psychiatryonline.org/article ... id=1676090
http://www.ncbi.nlm.nih.gov/pubmed/23558429

[fuKs]

Własnie o to chodzi w działaniu neuroleptyku żebyś nie był w stanie myśleć bo gdy myślisz wymyślasz takie twory .

źródło: http://www.nerwica.com

Po przeczytaniu x artukułów wysłuchaniu x doktorków i specjalistów dopiero teraz, po tym fragmencie postu dowiedziałem się jak działa psychotrop.

[Unknown]

http://www.madinamerica.com/wp-content/ ... reason.pdf - dr. Andreasen kosztowała 38mln dolców...

[Niemamnie]

Leki powodują trwałe zmiany w mózgu, szczególnie w płacie czołowym.

[Unknown]

http://www.madinamerica.com/wp-content/ ... reason.pdf

"Conclusions: Extended periods of relapse may have a negative effect on brain integrity in schizophrenia, suggesting the importance of implementing proactive measures that may prevent relapse and improve treatment adherence. By examining the relative balance of effects, that is, relapse duration versus antipsychotic treatment intensity, this study sheds light on a troublesome dilemma that clinicians face. Relapse prevention is important, but it should be sustained using the lowest possible medication dosages that will control symptoms."

A Close Look at Andreasen et al.’s Advice to Increase the Dosage of Antipsychotics to Prevent Brain Volume Reduction - http://www.madinamerica.com/2013/06/a-c ... reduction/

Actually, I thought that drugs that block dopamine receptors, in the end, INCREASE dopamine production, because the dopamine isn’t doing the job it otherwise would. And this is also how they got the statistics that dopamine hyperactivity has to do with schizophrenia, because when you drug people with dopamine receptor blocking “medications” in the end you get dopamine hyperactivity, because the brain tries to compensate for the dopamine that’s not allowed to work, and makes more.

This then would explain the following also: “A number of investigators have found elevations in free radicals and lower levels of antioxidants in persons exhibiting psychosis who were not taking medications (Arvindakshan, et al., 2003; Khan et al., 2002; Li et al., 2011; Mahadik et al., 1998; Owe-Larsson, et al., 2011; Yao et al., 1998; Zhang et al., 2009). Furthermore, the level of oxidative stress in unmedicated persons with psychosis correlates with level of negative symptoms (Arvindakshan et al., 2003; Li, Zheng, Xiu 2011) and positive symptoms (Mahadik et al., 1998; Zhang et al., 2009). ” because we aren’t talking about “unmedicated” people. We are talking about people who have been medicated, had their brain try to compensate for the blocking of dopamine, and then when they try to get off of medications which make them experience their thoughts as further and further away, and muted; they get dopamine hyperactivity, confusion, stress and then these free radicals. Which easily could come from the stress (both physical and emotional) rather than anything to do with “schizophrenia,” “psychosis,” etc..

Also, you’re grouping together “psychosis” or “psychotic symptoms” from cocaine use, and psychosis from true emotional trauma. Those are two very different things. And it’s been shown that people with psychosis from emotional trauma do the best when NOT medicated in any way, but receive support, and are put in an environment that’s not alarmed by responses they have, and they can let go of their fear rather than be further assaulted by the indoctrinated around them.

And it’s completely true that the whole connection with symptoms and their cause is obfuscated here. To add to that dopamine receptor blocking medications increase dopamine production, they may suppress expression of what the psychiatrist calls symptoms. Ad these are called symptoms for lack of insight into what a patients emotional experiences have been. When these “symptoms” are only magnified because of “treatment” this still makes no correlation with brain reduction and “schizophrenia” itself. It’s the increase in dopamine the dopamine receptor blocking drugs caused that correlates with “symptoms” and with further suppression of a person’s expression. And this expression has been shown to be allowed to heal when allowed in other settings not using these “medications.” This whole talk about brain reduction makes no sense. It’s trying to make out that the increase in symptoms caused by the lack of emotional help and the implimentation of medications, which caused increase in dopamine production (and has been used to pin on the “disease” rather than the medications) that this then is used to correlate with the brain reduction that statistically clearly correlates with the medications.

You would have to allow a person who is having “symptoms” to be given the help that has been shown to be more effective than medications, and then see whether this prevents brain reduction. That, they’re not interested in, to begin with. They’d have to try a different approach than the biological model they have yet to prove is actually biological. And so they talk about brain reduction correlating with one thing, when it clearly (and scientifically) correlates with the other, which is their unproven biological model, which promotes these “medications.”

[JudasHonor]

Translate please
You can put it on some kind of google translator or something like that and paste it here.

[Unknown]

Chyba nie myślisz, że będę tłumaczyć każdy artykuł jaki się pojawi na ten temat czy wyniki badań o oczywistej szkodliwości psychotropów. W skrócie: neuroleptyki powodują neurodegradacje i powinny być stosowane w jak najmniejszych skutecznych dawkach i tylko jeśli to konieczne.

google translator or something

Good luck with that.

[moi]

Schizo, też prosiłabym, żebyś umieszczała tłumaczenia, zamiast obcojęzyczynych cytatów. To jednak forum polskojęzyczne.

Pozdrawiam.m.

[Petruccio]

To chyba nie wina Schizo, jeśli takie informacje są podawane tylko na stronach obcojęzycznych. A chyba każdy umie korzystać z Tłumacza Google? Polecam.

http://translate.google.pl/

[Unknown]

Antipsychotics and Brain Shrinkage: An Update http://www.madinamerica.com/2013/06/ant ... an-update/

Evidence that antipsychotics cause brain shrinkage has been accumulating over the last few years, but the psychiatric research establishment is finding its own results difficult to swallow. A new paper by a group of American researchers once again tries to ‘blame the disease,’ a time-honoured tactic for diverting attention from the nasty and dangerous effects of some psychiatric treatments.

[...]

So Andreasen’s group have found strong evidence of an antipsychotic-induced effect, which they have replicated in two analyses now. The predictive value of the severity of symptoms, on the other hand (which is essentially what relapse appears to define) is weak in the initial analysis, and in neither analysis was it clearly differentiated from drug-induced effects.

These researchers seem determined to prove that ‘schizophrenia’ causes brain shrinkage, although their data simply cannot establish this, as none of their subjects seem to have gone without drug treatment for any significant length of time. So even though their recent analysis once again confirms the damaging effects of antipsychotics, they conclude that the results demonstrate the need to make sure patients take, and do not stop, their antipsychotic medication. The only concession made to the antipsychotic-induced changes revealed is the suggestion that low doses of antipsychotics should be used where possible.

Yet other prominent psychiatric researchers have now abandoned the idea that schizophrenia is a progressive, neurodegenerative condition, and do not consider that Andreasen’s study provides evidence of this.(10) Bizarrely, Nancy Andreasen is a co-author of a recently published meta-analysis which combines results of 30 studies of brain volume over time, which clearly confirms the association between antipsychotic treatment and brain shrinkage (specifically the grey matter) and finds no relationship with severity of symptoms or duration of the underlying condition.(11)

What should antipsychotic users and their families and carers make of this research? Obviously it sounds frightening and worrying, but the first thing to stress is that the reductions in brain volume that are detected in these MRI studies are small, and it is not certain that changes of this sort have any functional implications. We do not yet know whether these changes are reversible or not. Of course the value of antipsychotics has been much debated on this site and elsewhere, and their utility almost certainly depends on the particular circumstances of each individual user, so it is impossible to issue any blanket advice. If people are worried, they need to discuss the pros and cons of continuing to take antipsychotic treatment with their prescriber, bearing in mind the difficulties that are associated with coming off these drugs.(12) People should not stop drug treatment suddenly, especially if they have been taking it for a long time.

People need to know about this research because it indicates that antipsychotics are not the innocuous substances that they have frequently been portrayed as. We still have no conclusive evidence that the disorders labeled as schizophrenia or psychosis are associated with any underlying abnormalities of the brain, but we do have strong evidence that the drugs we use to treat these conditions cause brain changes. This does not mean that taking antipsychotics is not sometimes useful and worthwhile, despite these effects, but it does mean we have to be very cautious indeed about using them.

[moi]

Widzę, że zupełnie zignorowałaś nasze prośby. Szkoda, bo Twoje posty stają się nieprzydatne.

Pozdrawiam.m.

[Petruccio]

Dobrze by było, gdyby chociaż zarys był po polsku - żeby zachęcić czytelnika do przetłumaczenia reszty ;-)

[JudasHonor]

Każdy sam sobie zapracowuje na wartość swoich wypowiedzi, niech robi więc tak jak uważa.